Conn's Syndrome FAQ
What causes primary hyperaldosteronism?
It's often due to a benign adrenal gland tumor or hyperplasia, leading to excessive aldosterone production.
What are common symptoms?
High blood pressure, muscle weakness, fatigue, and low potassium levels (hypokalemia).
How is primary hyperaldosteronism diagnosed?
Through blood tests, urine tests, and imaging studies to assess aldosterone and potassium levels.
Is surgery necessary for treatment?
Possibly, if a tumor is present; otherwise, medications like spironolactone may be prescribed.
Can it be cured?
If caused by a tumor, surgical removal can cure it; otherwise, it's managed with medication.
How does it affect blood pressure?
Excess aldosterone increases sodium retention, leading to high blood pressure.
What dietary changes help?
Reducing sodium intake and increasing potassium-rich foods can help manage symptoms.
Can it lead to complications?
Untreated, it can cause heart disease, stroke, or kidney damage due to prolonged high blood pressure.
Is it hereditary?
Rarely; most cases are acquired, though genetic forms exist (e.g., familial hyperaldosteronism).
How often should I monitor my potassium levels?
Regularly, as low potassium (hypokalemia) is common; frequency depends on your doctor's advice.
Can I manage it with lifestyle changes alone?
Unlikely, as it typically requires medication or surgery to control aldosterone levels.
What medications are commonly used?
Potassium-sparing diuretics like spironolactone or eplerenone.
How does it differ from secondary hyperaldosteronism?
Primary is due to adrenal gland issues, while secondary is caused by external factors like heart failure.
Can it cause headaches?
Yes, as high blood pressure can lead to headaches, though not everyone experiences them.
What is the prognosis with treatment?
Generally good, with effective management of blood pressure and potassium levels reducing complications.
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